DIABETES – DAMAGES AND TREATMENTS by Everlon Cid Rigobelo

By Everlon Cid Rigobelo

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Thus, fatty acid oxygenation is required to provide substrate for gluconeogenesis and ketogenesis. Ketones, the byproduct of fatty acid metabolism, are found in urine and represent the starved state. Increased glucose use (eg, hyperthermia, polycythemia, sepsis, growth hormone deficiency), decreased glycogenolysis, gluconeogenesis, or use of alternate fuels (eg, inborn errors of metabolism, adrenal insufficiency) (Cranmer, 2009, Guideline, 2004). , 2004). , 2004, Stanley, 1997). , 2009). Infants of mothers with diabetes also have high insulin levels after birth due to the high glucose exposure in utero; the poorer the glucose control during pregnancy, the greater the likelihood of hyperinsulinism in the infant (Stanley, 1997).

2001) Exercise induced hypoglycaemic hyperinsulinism. N. (2002) Experience with intravenous glucagon infusions as a treatment for resistant neonatal hypoglycaemia. A. E. (2002) Acarbose treatment of postprandial hypoglycemia in children after Nissen fundoplication. (2007) Hypoglycemia and growth failure due to inhaled corticosteroids. L. (2007) Diagnosis and management of hypoglycaemia beyond the neonatal period. Q. (1999) Autoimmune hypoglycemia. J. (2002) Fatty acid oxidation disorders-. H. (2001) Mitochondrial fatty acid oxidation disorders.

2003). , 2008). Avoid hyperglycemia evoking prompt insulin release, which may produce rebound hypoglycemia. SGA infants and those with maternal toxemia or perinatal asphyxia require dextrose IV infusion rates >20 mg/kg/min to control levels (Halamek and Stevenson, 1998). Treatment may be necessary for 2-4 week . Diazoxide Aim to increase blood glucose by inhibiting pancreatic insulin release, and possibly through an extrapancreatic effect. , 2008, Shirland, 2001). , 1972). Glucagon (Glucagon Emergency Kit) May be used to treat hypoglycemia secondary to hyperinsulinemia and administered to patients without initial IV access.

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